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bipolar emg signals  (PLUX Biosignals SA)


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    Structured Review

    PLUX Biosignals SA bipolar emg signals
    The original <t>EMG</t> <t>signals</t> of the MRF, MVM, MBF-CL, and MS muscles from two representative subjects participating in Experiment I ( A ) and Experiment II ( B ), respectively.
    Bipolar Emg Signals, supplied by PLUX Biosignals SA, used in various techniques. Bioz Stars score: 93/100, based on 123 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/bipolar+emg+signals/Electromyography+(EMG)+Sensor/pmc12389886-108-0-9
    Average 93 stars, based on 123 article reviews
    bipolar emg signals - by Bioz Stars, 2026-09
    93/100 stars

    Images

    1) Product Images from "Pain State Classification of Stiff Knee Joint Using Electromyogram for Robot-Based Post-Fracture Rehabilitation Training"

    Article Title: Pain State Classification of Stiff Knee Joint Using Electromyogram for Robot-Based Post-Fracture Rehabilitation Training

    Journal: Sensors (Basel, Switzerland)

    doi: 10.3390/s25165142

    The original EMG signals of the MRF, MVM, MBF-CL, and MS muscles from two representative subjects participating in Experiment I ( A ) and Experiment II ( B ), respectively.
    Figure Legend Snippet: The original EMG signals of the MRF, MVM, MBF-CL, and MS muscles from two representative subjects participating in Experiment I ( A ) and Experiment II ( B ), respectively.

    Techniques Used: Muscles

    Related Articles

    Amplification:

    Article Title: Pain State Classification of Stiff Knee Joint Using Electromyogram for Robot-Based Post-Fracture Rehabilitation Training
    Article Snippet: .. Bipolar EMG signals were amplified and acquired using a Biosignalsplux system (PLUX, Lisboa, Portugal) with a sampling rate of 2000 Hz. ..

    Article Title: Comparison of the activation level in the sensorimotor cortex between motor point and proximal nerve bundle electrical stimulation.
    Article Snippet: .. Bipolar EMG signals were amplified using the Bluetooth 2.0 + EDR system (Biosignalsplux professional, PLUX, Portugal) with a gain of 1000 and a pass band of 25–500 Hz, and sampled at 2000 Hz. ..

    Sampling:

    Article Title: Pain State Classification of Stiff Knee Joint Using Electromyogram for Robot-Based Post-Fracture Rehabilitation Training
    Article Snippet: .. Bipolar EMG signals were amplified and acquired using a Biosignalsplux system (PLUX, Lisboa, Portugal) with a sampling rate of 2000 Hz. ..



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    PLUX Biosignals SA bipolar emg signals
    The original <t>EMG</t> <t>signals</t> of the MRF, MVM, MBF-CL, and MS muscles from two representative subjects participating in Experiment I ( A ) and Experiment II ( B ), respectively.
    Bipolar Emg Signals, supplied by PLUX Biosignals SA, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Electromyographic activity in light-activated contractions. a Example <t>EMG</t> recording with simultaneous jaw tracking during optogenetic activation of jaw muscles. Top: 5 ms light pulses of varying light intensity; middle: recorded EMG activity (black) and smoothened EMG signal (red); bottom: jaw movements (jaw closing) captured with high-speed videography. b-d Relationship between light intensity and peak amplitude of smoothened EMG signal ( b ), between light intensity and latency to peak ( c ), and between sum of smoothened EMG signal and maximum jaw displacement ( d ) in 5 ms (blue), 50 ms (red), 100 ms (yellow) light pulses and 1 Hz sinusoid stimuli (purple) (n = 5 trials per stimulus, 4 recordings, 2 mice). Mean ± s.e.m. (error shade). e, f Spatial profile of peak amplitude of smoothened EMG signal ( e ) and latency to peak ( f ) for single trial at 1mW/mm 2 illumination intensity shows decaying peak amplitude and increasing latency with distance from center of illumination. g Space constant of peak amplitude does not show clear dependence on light intensity (n = 5 trials, 1 recording, 1 mouse). h-l Properties of <t>EMG</t> <t>signals</t> during slowly ramping optogenetic stimuli. h Representative recording shows initial EMG activity followed by suppression without overt jaw movements. Top: light intensity, middle: EMG signal, bottom: jaw movement. i Sparse waveforms are observed in the EMG signals during slow ramp ( h : shaded insets). j Spike waveforms colored with increasing opacity based on light intensity shows decreasing amplitude with preserved shape of waveform. k Amplitude of spike waveforms decrease with light intensity. l Inter-spike interval (ISI) decreases to a plateau at 16 ms with increasing light intensity.
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    Electromyographic activity in light-activated contractions. a Example <t>EMG</t> recording with simultaneous jaw tracking during optogenetic activation of jaw muscles. Top: 5 ms light pulses of varying light intensity; middle: recorded EMG activity (black) and smoothened EMG signal (red); bottom: jaw movements (jaw closing) captured with high-speed videography. b-d Relationship between light intensity and peak amplitude of smoothened EMG signal ( b ), between light intensity and latency to peak ( c ), and between sum of smoothened EMG signal and maximum jaw displacement ( d ) in 5 ms (blue), 50 ms (red), 100 ms (yellow) light pulses and 1 Hz sinusoid stimuli (purple) (n = 5 trials per stimulus, 4 recordings, 2 mice). Mean ± s.e.m. (error shade). e, f Spatial profile of peak amplitude of smoothened EMG signal ( e ) and latency to peak ( f ) for single trial at 1mW/mm 2 illumination intensity shows decaying peak amplitude and increasing latency with distance from center of illumination. g Space constant of peak amplitude does not show clear dependence on light intensity (n = 5 trials, 1 recording, 1 mouse). h-l Properties of <t>EMG</t> <t>signals</t> during slowly ramping optogenetic stimuli. h Representative recording shows initial EMG activity followed by suppression without overt jaw movements. Top: light intensity, middle: EMG signal, bottom: jaw movement. i Sparse waveforms are observed in the EMG signals during slow ramp ( h : shaded insets). j Spike waveforms colored with increasing opacity based on light intensity shows decreasing amplitude with preserved shape of waveform. k Amplitude of spike waveforms decrease with light intensity. l Inter-spike interval (ISI) decreases to a plateau at 16 ms with increasing light intensity.
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    Electromyographic activity in light-activated contractions. a Example <t>EMG</t> recording with simultaneous jaw tracking during optogenetic activation of jaw muscles. Top: 5 ms light pulses of varying light intensity; middle: recorded EMG activity (black) and smoothened EMG signal (red); bottom: jaw movements (jaw closing) captured with high-speed videography. b-d Relationship between light intensity and peak amplitude of smoothened EMG signal ( b ), between light intensity and latency to peak ( c ), and between sum of smoothened EMG signal and maximum jaw displacement ( d ) in 5 ms (blue), 50 ms (red), 100 ms (yellow) light pulses and 1 Hz sinusoid stimuli (purple) (n = 5 trials per stimulus, 4 recordings, 2 mice). Mean ± s.e.m. (error shade). e, f Spatial profile of peak amplitude of smoothened EMG signal ( e ) and latency to peak ( f ) for single trial at 1mW/mm 2 illumination intensity shows decaying peak amplitude and increasing latency with distance from center of illumination. g Space constant of peak amplitude does not show clear dependence on light intensity (n = 5 trials, 1 recording, 1 mouse). h-l Properties of <t>EMG</t> <t>signals</t> during slowly ramping optogenetic stimuli. h Representative recording shows initial EMG activity followed by suppression without overt jaw movements. Top: light intensity, middle: EMG signal, bottom: jaw movement. i Sparse waveforms are observed in the EMG signals during slow ramp ( h : shaded insets). j Spike waveforms colored with increasing opacity based on light intensity shows decreasing amplitude with preserved shape of waveform. k Amplitude of spike waveforms decrease with light intensity. l Inter-spike interval (ISI) decreases to a plateau at 16 ms with increasing light intensity.
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    Electromyographic activity in light-activated contractions. a Example <t>EMG</t> recording with simultaneous jaw tracking during optogenetic activation of jaw muscles. Top: 5 ms light pulses of varying light intensity; middle: recorded EMG activity (black) and smoothened EMG signal (red); bottom: jaw movements (jaw closing) captured with high-speed videography. b-d Relationship between light intensity and peak amplitude of smoothened EMG signal ( b ), between light intensity and latency to peak ( c ), and between sum of smoothened EMG signal and maximum jaw displacement ( d ) in 5 ms (blue), 50 ms (red), 100 ms (yellow) light pulses and 1 Hz sinusoid stimuli (purple) (n = 5 trials per stimulus, 4 recordings, 2 mice). Mean ± s.e.m. (error shade). e, f Spatial profile of peak amplitude of smoothened EMG signal ( e ) and latency to peak ( f ) for single trial at 1mW/mm 2 illumination intensity shows decaying peak amplitude and increasing latency with distance from center of illumination. g Space constant of peak amplitude does not show clear dependence on light intensity (n = 5 trials, 1 recording, 1 mouse). h-l Properties of <t>EMG</t> <t>signals</t> during slowly ramping optogenetic stimuli. h Representative recording shows initial EMG activity followed by suppression without overt jaw movements. Top: light intensity, middle: EMG signal, bottom: jaw movement. i Sparse waveforms are observed in the EMG signals during slow ramp ( h : shaded insets). j Spike waveforms colored with increasing opacity based on light intensity shows decreasing amplitude with preserved shape of waveform. k Amplitude of spike waveforms decrease with light intensity. l Inter-spike interval (ISI) decreases to a plateau at 16 ms with increasing light intensity.
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    Electromyographic activity in light-activated contractions. a Example <t>EMG</t> recording with simultaneous jaw tracking during optogenetic activation of jaw muscles. Top: 5 ms light pulses of varying light intensity; middle: recorded EMG activity (black) and smoothened EMG signal (red); bottom: jaw movements (jaw closing) captured with high-speed videography. b-d Relationship between light intensity and peak amplitude of smoothened EMG signal ( b ), between light intensity and latency to peak ( c ), and between sum of smoothened EMG signal and maximum jaw displacement ( d ) in 5 ms (blue), 50 ms (red), 100 ms (yellow) light pulses and 1 Hz sinusoid stimuli (purple) (n = 5 trials per stimulus, 4 recordings, 2 mice). Mean ± s.e.m. (error shade). e, f Spatial profile of peak amplitude of smoothened EMG signal ( e ) and latency to peak ( f ) for single trial at 1mW/mm 2 illumination intensity shows decaying peak amplitude and increasing latency with distance from center of illumination. g Space constant of peak amplitude does not show clear dependence on light intensity (n = 5 trials, 1 recording, 1 mouse). h-l Properties of <t>EMG</t> <t>signals</t> during slowly ramping optogenetic stimuli. h Representative recording shows initial EMG activity followed by suppression without overt jaw movements. Top: light intensity, middle: EMG signal, bottom: jaw movement. i Sparse waveforms are observed in the EMG signals during slow ramp ( h : shaded insets). j Spike waveforms colored with increasing opacity based on light intensity shows decreasing amplitude with preserved shape of waveform. k Amplitude of spike waveforms decrease with light intensity. l Inter-spike interval (ISI) decreases to a plateau at 16 ms with increasing light intensity.
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    Electromyographic activity in light-activated contractions. a Example <t>EMG</t> recording with simultaneous jaw tracking during optogenetic activation of jaw muscles. Top: 5 ms light pulses of varying light intensity; middle: recorded EMG activity (black) and smoothened EMG signal (red); bottom: jaw movements (jaw closing) captured with high-speed videography. b-d Relationship between light intensity and peak amplitude of smoothened EMG signal ( b ), between light intensity and latency to peak ( c ), and between sum of smoothened EMG signal and maximum jaw displacement ( d ) in 5 ms (blue), 50 ms (red), 100 ms (yellow) light pulses and 1 Hz sinusoid stimuli (purple) (n = 5 trials per stimulus, 4 recordings, 2 mice). Mean ± s.e.m. (error shade). e, f Spatial profile of peak amplitude of smoothened EMG signal ( e ) and latency to peak ( f ) for single trial at 1mW/mm 2 illumination intensity shows decaying peak amplitude and increasing latency with distance from center of illumination. g Space constant of peak amplitude does not show clear dependence on light intensity (n = 5 trials, 1 recording, 1 mouse). h-l Properties of <t>EMG</t> <t>signals</t> during slowly ramping optogenetic stimuli. h Representative recording shows initial EMG activity followed by suppression without overt jaw movements. Top: light intensity, middle: EMG signal, bottom: jaw movement. i Sparse waveforms are observed in the EMG signals during slow ramp ( h : shaded insets). j Spike waveforms colored with increasing opacity based on light intensity shows decreasing amplitude with preserved shape of waveform. k Amplitude of spike waveforms decrease with light intensity. l Inter-spike interval (ISI) decreases to a plateau at 16 ms with increasing light intensity.
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    Electromyographic activity in light-activated contractions. a Example <t>EMG</t> recording with simultaneous jaw tracking during optogenetic activation of jaw muscles. Top: 5 ms light pulses of varying light intensity; middle: recorded EMG activity (black) and smoothened EMG signal (red); bottom: jaw movements (jaw closing) captured with high-speed videography. b-d Relationship between light intensity and peak amplitude of smoothened EMG signal ( b ), between light intensity and latency to peak ( c ), and between sum of smoothened EMG signal and maximum jaw displacement ( d ) in 5 ms (blue), 50 ms (red), 100 ms (yellow) light pulses and 1 Hz sinusoid stimuli (purple) (n = 5 trials per stimulus, 4 recordings, 2 mice). Mean ± s.e.m. (error shade). e, f Spatial profile of peak amplitude of smoothened EMG signal ( e ) and latency to peak ( f ) for single trial at 1mW/mm 2 illumination intensity shows decaying peak amplitude and increasing latency with distance from center of illumination. g Space constant of peak amplitude does not show clear dependence on light intensity (n = 5 trials, 1 recording, 1 mouse). h-l Properties of <t>EMG</t> <t>signals</t> during slowly ramping optogenetic stimuli. h Representative recording shows initial EMG activity followed by suppression without overt jaw movements. Top: light intensity, middle: EMG signal, bottom: jaw movement. i Sparse waveforms are observed in the EMG signals during slow ramp ( h : shaded insets). j Spike waveforms colored with increasing opacity based on light intensity shows decreasing amplitude with preserved shape of waveform. k Amplitude of spike waveforms decrease with light intensity. l Inter-spike interval (ISI) decreases to a plateau at 16 ms with increasing light intensity.
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    Electromyographic activity in light-activated contractions. a Example <t>EMG</t> recording with simultaneous jaw tracking during optogenetic activation of jaw muscles. Top: 5 ms light pulses of varying light intensity; middle: recorded EMG activity (black) and smoothened EMG signal (red); bottom: jaw movements (jaw closing) captured with high-speed videography. b-d Relationship between light intensity and peak amplitude of smoothened EMG signal ( b ), between light intensity and latency to peak ( c ), and between sum of smoothened EMG signal and maximum jaw displacement ( d ) in 5 ms (blue), 50 ms (red), 100 ms (yellow) light pulses and 1 Hz sinusoid stimuli (purple) (n = 5 trials per stimulus, 4 recordings, 2 mice). Mean ± s.e.m. (error shade). e, f Spatial profile of peak amplitude of smoothened EMG signal ( e ) and latency to peak ( f ) for single trial at 1mW/mm 2 illumination intensity shows decaying peak amplitude and increasing latency with distance from center of illumination. g Space constant of peak amplitude does not show clear dependence on light intensity (n = 5 trials, 1 recording, 1 mouse). h-l Properties of <t>EMG</t> <t>signals</t> during slowly ramping optogenetic stimuli. h Representative recording shows initial EMG activity followed by suppression without overt jaw movements. Top: light intensity, middle: EMG signal, bottom: jaw movement. i Sparse waveforms are observed in the EMG signals during slow ramp ( h : shaded insets). j Spike waveforms colored with increasing opacity based on light intensity shows decreasing amplitude with preserved shape of waveform. k Amplitude of spike waveforms decrease with light intensity. l Inter-spike interval (ISI) decreases to a plateau at 16 ms with increasing light intensity.
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    Image Search Results


    The original EMG signals of the MRF, MVM, MBF-CL, and MS muscles from two representative subjects participating in Experiment I ( A ) and Experiment II ( B ), respectively.

    Journal: Sensors (Basel, Switzerland)

    Article Title: Pain State Classification of Stiff Knee Joint Using Electromyogram for Robot-Based Post-Fracture Rehabilitation Training

    doi: 10.3390/s25165142

    Figure Lengend Snippet: The original EMG signals of the MRF, MVM, MBF-CL, and MS muscles from two representative subjects participating in Experiment I ( A ) and Experiment II ( B ), respectively.

    Article Snippet: Bipolar EMG signals were amplified and acquired using a Biosignalsplux system (PLUX, Lisboa, Portugal) with a sampling rate of 2000 Hz.

    Techniques: Muscles

    Electromyographic activity in light-activated contractions. a Example EMG recording with simultaneous jaw tracking during optogenetic activation of jaw muscles. Top: 5 ms light pulses of varying light intensity; middle: recorded EMG activity (black) and smoothened EMG signal (red); bottom: jaw movements (jaw closing) captured with high-speed videography. b-d Relationship between light intensity and peak amplitude of smoothened EMG signal ( b ), between light intensity and latency to peak ( c ), and between sum of smoothened EMG signal and maximum jaw displacement ( d ) in 5 ms (blue), 50 ms (red), 100 ms (yellow) light pulses and 1 Hz sinusoid stimuli (purple) (n = 5 trials per stimulus, 4 recordings, 2 mice). Mean ± s.e.m. (error shade). e, f Spatial profile of peak amplitude of smoothened EMG signal ( e ) and latency to peak ( f ) for single trial at 1mW/mm 2 illumination intensity shows decaying peak amplitude and increasing latency with distance from center of illumination. g Space constant of peak amplitude does not show clear dependence on light intensity (n = 5 trials, 1 recording, 1 mouse). h-l Properties of EMG signals during slowly ramping optogenetic stimuli. h Representative recording shows initial EMG activity followed by suppression without overt jaw movements. Top: light intensity, middle: EMG signal, bottom: jaw movement. i Sparse waveforms are observed in the EMG signals during slow ramp ( h : shaded insets). j Spike waveforms colored with increasing opacity based on light intensity shows decreasing amplitude with preserved shape of waveform. k Amplitude of spike waveforms decrease with light intensity. l Inter-spike interval (ISI) decreases to a plateau at 16 ms with increasing light intensity.

    Journal: bioRxiv

    Article Title: Myo-optogenetics: optogenetic stimulation and electrical recording in skeletal muscles

    doi: 10.1101/2024.06.21.600113

    Figure Lengend Snippet: Electromyographic activity in light-activated contractions. a Example EMG recording with simultaneous jaw tracking during optogenetic activation of jaw muscles. Top: 5 ms light pulses of varying light intensity; middle: recorded EMG activity (black) and smoothened EMG signal (red); bottom: jaw movements (jaw closing) captured with high-speed videography. b-d Relationship between light intensity and peak amplitude of smoothened EMG signal ( b ), between light intensity and latency to peak ( c ), and between sum of smoothened EMG signal and maximum jaw displacement ( d ) in 5 ms (blue), 50 ms (red), 100 ms (yellow) light pulses and 1 Hz sinusoid stimuli (purple) (n = 5 trials per stimulus, 4 recordings, 2 mice). Mean ± s.e.m. (error shade). e, f Spatial profile of peak amplitude of smoothened EMG signal ( e ) and latency to peak ( f ) for single trial at 1mW/mm 2 illumination intensity shows decaying peak amplitude and increasing latency with distance from center of illumination. g Space constant of peak amplitude does not show clear dependence on light intensity (n = 5 trials, 1 recording, 1 mouse). h-l Properties of EMG signals during slowly ramping optogenetic stimuli. h Representative recording shows initial EMG activity followed by suppression without overt jaw movements. Top: light intensity, middle: EMG signal, bottom: jaw movement. i Sparse waveforms are observed in the EMG signals during slow ramp ( h : shaded insets). j Spike waveforms colored with increasing opacity based on light intensity shows decreasing amplitude with preserved shape of waveform. k Amplitude of spike waveforms decrease with light intensity. l Inter-spike interval (ISI) decreases to a plateau at 16 ms with increasing light intensity.

    Article Snippet: We recorded EMG signals in 16 channel bipolar headstage (Intan RHD2216) and band-passed the signals between 300-9000 Hz.

    Techniques: Activity Assay, Activation Assay, Muscles

    Example electromyography activity during optogenetically evoked and spontaneous movements. a Schematic depicting Myomatrix EMG recording in temporalis with optogenetic stimuli. A grid of electrode contacts in a Myomatrix array allows 16 bipolar recording sites. b Example EMG signals from temporalis muscle at 16 recording sites during light pulse (1mW/mm 2 , 5 ms). Amplitude of EMG activity decayswith distance from the center of optogenetic stimulation. Vertical scale bar: 0.2 mV; horizontal scale bar: 5 ms. c Example spotaneous EMG activity (right) in temporalis muscle of Emx1 Cre/+ ; Rosa26LSL-ChR2-EYFP/+ mice (left). Motor unit activity is characterized by sparse, well-defined waveforms in contrast to dense activity during optogenetic stimulation. d Example EMG recording (right) in temporalis muscle of wild-type (C57BL/6) mouse during light pulse (3.2 mW/mm 2 , 5ms) (left). Muscle fibers without ChR2 do not show electrical activity in response to optogenetic stimulation.

    Journal: bioRxiv

    Article Title: Myo-optogenetics: optogenetic stimulation and electrical recording in skeletal muscles

    doi: 10.1101/2024.06.21.600113

    Figure Lengend Snippet: Example electromyography activity during optogenetically evoked and spontaneous movements. a Schematic depicting Myomatrix EMG recording in temporalis with optogenetic stimuli. A grid of electrode contacts in a Myomatrix array allows 16 bipolar recording sites. b Example EMG signals from temporalis muscle at 16 recording sites during light pulse (1mW/mm 2 , 5 ms). Amplitude of EMG activity decayswith distance from the center of optogenetic stimulation. Vertical scale bar: 0.2 mV; horizontal scale bar: 5 ms. c Example spotaneous EMG activity (right) in temporalis muscle of Emx1 Cre/+ ; Rosa26LSL-ChR2-EYFP/+ mice (left). Motor unit activity is characterized by sparse, well-defined waveforms in contrast to dense activity during optogenetic stimulation. d Example EMG recording (right) in temporalis muscle of wild-type (C57BL/6) mouse during light pulse (3.2 mW/mm 2 , 5ms) (left). Muscle fibers without ChR2 do not show electrical activity in response to optogenetic stimulation.

    Article Snippet: We recorded EMG signals in 16 channel bipolar headstage (Intan RHD2216) and band-passed the signals between 300-9000 Hz.

    Techniques: Activity Assay